Only in Titles

           Search results for: Elisa kits   

paperclip

#31958332   // Save this To Up

High seroprevalence of hepatitis E among pigs suggests an animal reservoir in Cameroon.

Hepatitis E virus (HEV) is one of the most prevalent cause of acute hepatitis in humans worldwide. The risk of HEV transmission is not limited only to spread from human to human but the infection can also spread from animals to humans, especially from the domestic pigs. Despite mounting evidence regarding the zoonotic potential of porcine HEV infection, there are limited data on its prevalence in pigs in the sub-Sahara Africa region. Therefore, the present study aimed to determine the seroprevalence of HEV antibodies among pigs in two Cameroonian regions.

2489 related Products with: High seroprevalence of hepatitis E among pigs suggests an animal reservoir in Cameroon.

Alkaline Phospatase (ALP) ErbB Her Signaling Phosph Proteins and Antibodies H GPCR Signaling to MAPK ER Goat Anti-Human ERK2 MAPK Human interleukin 2(IL-2) CDC6 & E2F1 Protein Prote NF-kB Phospho-Specific Ar Goat Anti-Human E2F7, (in Goat Anti-Human Ezrin vil Angiogenesis (Human) Anti SMURF1 & ECSIT Protein Pr

Related Pathways

paperclip

#31957860   // Save this To Up

Effect of rosiglitazone on myocardial injury in septic rats through NF-κB pathway.

To explore the effect of rosiglitazone on myocardial injury in septic rats through the nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB) pathway.

2800 related Products with: Effect of rosiglitazone on myocardial injury in septic rats through NF-κB pathway.

AMPK Signaling Phospho-Sp ERK Signaling Phospho-Spe mTOR Signalign Phospho-Sp Goat Anti-Human Tissue Fa Signal transduction antib Cytoskeleton Phospho-Spec Jak Stat Phospho-Specific T-Cell Receptor Signaling Apoptosis antibody array Cell Cycle Phospho-Specif IGF-1R Signaling Phospho- NF-kB II Phospho-Specific

Related Pathways

paperclip

#31957846   // Save this To Up

S100B as a new fecal biomarker of inflammatory bowel diseases.

S100 proteins are demonstrated to exert a protective role in the gastrointestinal tract. In the present study, we investigated whether S100B protein, that is typically expressed by enteroglial cells, is detectable in feces and could be a useful noninvasive indicator of gut chronic inflammation.

2287 related Products with: S100B as a new fecal biomarker of inflammatory bowel diseases.

ENZYMATIC ASSAY KITS (CH ENZYMATIC ASSAY KITS (CH ENZYMATIC ASSAY KITS (CH ENZYMATIC ASSAY KITS (CH ASF1A Antibody Cell Meter™ Caspase 9 A Caspase-3 Substrate DEVD- Quick Cell Proliferation anti Asparaginase Screen Quest™ Fluo 8 Me Caspase-5 Substrate WEHD- Lactate Assay Kit II

Related Pathways

paperclip

#31957703   // Save this To Up

Dexmedetomidine alleviates hepatic injury via the inhibition of oxidative stress and activation of the Nrf2/HO-1 signaling pathway.

Dexmedetomidine (Dex), frequently used as an effective sedative, was reported to play a critical role in the protection of multiple organs. However, its underlying mechanism of a putative protective effect on ischemia/reperfusion (I/R)-induced liver injury is still unclear. A hepatocyte injury model was established by treating WRL-68 cells with oxygen and glucose deprivation/reoxygenation (OGD/R). Enzyme Linked Immunosorbent Assay (ELISA) kits were used to determine the level of inflammatory factors (IL-6, IL-1β, and TNF-α), and oxidative stress indicators (ROS, MDA, GSH-Px, and SOD). MTT assay and flow cytometry analysis were used to determine the influence of Dex on cell viability and cell apoptosis. Expression of nuclear factor erythroid-derived 2- like 2 (Nrf2), HO-1, and apoptosis-related proteins (Bax, Bcl-2, caspase3, and caspase9) were detected by qRT-PCR and western blotting. Dex promoted cell viability and suppressed cell apoptosis in OGD/R-treated WRL-68 cells. Dex reduced TNF-α, IL-6, IL-1β, ROS, and MDA production, whereas it increased that of SOD and GSH-Px in OGD/R-treated WRL-68 cells. Moreover, Nrf2, HO-1, and Bcl-2 expression was upregulated, whereas, in contrast, transcripts for Bax, caspase3, and caspase9 were downregulated following Dex treatment under OGD/R. Knockdown of Nrf2 reversed the Dex effects on cell proliferation, apoptosis, and expression of TNF-α, IL-6, IL-1β, ROS, MDA, SOD, and GSH-Px. Dex protects WRL-68 cells against OGD/R-induced injury by inhibiting inflammation, oxidative stress, and cell apoptosis via the activation of Nrf2/HO-1 signaling pathway, suggesting that Dex may be a potential protector against hepatic injury.

1793 related Products with: Dexmedetomidine alleviates hepatic injury via the inhibition of oxidative stress and activation of the Nrf2/HO-1 signaling pathway.

Transcription factors: O TCP-1 theta antibody Sour Rabbit Anti-Rat Androgen FDA Standard Frozen Tissu Androstane 3a,17b diol Gl Wnt Signaling Pathway TCF ELISA TEK™ MBM Thermal Allergens, Phospholipase Recombinant Thermostable Thermostable TDG Kit *DIS ECOS 101 (DH5á) efficenc Rabbit anti PKC theta (Ab

Related Pathways

paperclip

#31949463   // Save this To Up

Zhujie Hewei Granules Ameliorated Reflux Esophagitis in Rats.

Gastroesophageal reflux disease (GERDs) is a common chronic digestive system disease, in which the symptoms of reflux esophagitis (RE) seriously affect the quality of life.

2022 related Products with: Zhujie Hewei Granules Ameliorated Reflux Esophagitis in Rats.

Breast cancer tissue arra ELISA Human , Interleukin Ovarian cancer test tissu Goat Anti-Human Dipeptidy Influenza A H3N2 Viral Ly Goat Anti-Human KPNA6, (i Caspase-2 Inhibitor Z-VDV JUP & CTNNB1 Protein Prot Rabbit Anti-NOS-2 iNOS Po CDK4 & CCND1 Protein Prot Colon adenocarcinoma tiss Interleukins Recombinant

Related Pathways

paperclip

#31948528   // Save this To Up

[Effect of asiaticoside on hyperoxia-induced bronchopulmonary dysplasia in neonatal rats and related mechanism].

To study the protective effect of asiaticoside against hyperoxia-induced bronchopulmonary dysplasia in neonatal rats based on the microRNA-155 (miR-155)/suppressor of cytokine signaling-1 (SOCS1) axis.

2812 related Products with: [Effect of asiaticoside on hyperoxia-induced bronchopulmonary dysplasia in neonatal rats and related mechanism].

Jurkat Cell Extract (Indu Mouse Epstein-Barr Virus Anti AICDA(Activation ind Rabbit Anti-FGF3 Oncogene Apoptosis Phospho-Specifi Anti AGO2 Human, Monoclon Jurkat Cell Extract (Indu TGF beta induced factor 2 GPCR Signaling to MAPK ER OxiSelect™ Cellular UV- Human Epstein-Barr Virus Anti-AICDA(Activation-ind

Related Pathways

paperclip

#31945029   // Save this To Up

Anti-Inflammatory and Antioxidant Effects of Acetyl-L-Carnitine on Atherosclerotic Rats.

BACKGROUND The purpose of the present study was to evaluate the regulatory effects of acetyl-L-carnitine (ALCAR) on atherosclerosis in Wister rats and to explore its anti-atherosclerotic mechanism. MATERIAL AND METHODS We randomly divided 32 Wister rats into 4 groups: a normal diet group (control group, n=8), a normal diet+ALCAR group (ALCAR group, n=8), an atherosclerosis group (AS group, n=8), and an atherosclerosis+ALCAR group (AS+ALCAR group, n=8). The serum lipid distribution, oxidative stress, inflammatory factors and adiponectin (APN) in the blood, and heart and aortic tissues were determined using the standard assay kits, xanthine oxidase method, and ELISA, respectively. HE staining was performed to observe aortic pathology structure change, and the level of angiotensin II (AngII) in the aorta was assessed using radioimmunoassay. In addition, real-time quantitative PCR and Western blot analysis were applied to detect the expression of iNOS, IL-1ß, TNF-alpha, and CRP in the aortic and heart tissues. RESULTS Compared with the AS group, the levels of serum TC, TG, LDL, and VLDL in rats decreased significantly, while HDL level significantly increased in the AS+ALCAR group. ALCAR administration enhanced the SOD and GSH-Px activities and decreased MDA activity. APN level was significantly elevated in the AS group, but ALCAR had no significant effect on APN. Further, ALCAR reduced the expressions of inflammation factors TNF-alpha, IL-1ß, iNOS, and CRP, and the concentration of AngII in serum, aortic, and heart tissues. CONCLUSIONS ALCAR can inhibit the expressions of inflammatory factors and antioxidation to suppress the development of atherosclerosis by adjusting blood lipid in the myocardium of AS rats.

2434 related Products with: Anti-Inflammatory and Antioxidant Effects of Acetyl-L-Carnitine on Atherosclerotic Rats.

Anti-Acetyl Cholinesteras Monoclonal Anti-acetyl- & 5α-N-Acetyl-2'H-androst- Anti Acetyl Histone H3 (A Anti-Conjugated Acetyl Sa Rabbit anti Androgen Rece Mouse Anti-HPV 16 Oncopro Rabbit Anti-Human Androge Anti acetyl Histone H3 (A Anti-acetyl Histone H3 (A Rabbit Anti-FGF3 Oncogene Anti-Acetyl Cholinesteras

Related Pathways

paperclip

#31940685   // Save this To Up

Comparison of the trometamol-balanced solution with two other crystalloid solutions for fluid resuscitation of a rat hemorrhagic model.

Currently, the optimal resuscitation fluid remains debatable. Therefore, in the present study, we designed a trometamol-balanced solution (TBS) for use as a resuscitation fluid for hemorrhagic shock. Hemorrhagic shock was induced in 18 male Wistar-Kyoto rats, which were assigned to normal saline (NS), Ringer's solution (RS), and TBS groups. During the hemorrhagic state, their hemodynamic parameters were recorded using an Abbott i-STAT analyzer with the CG4+ cartridge (for pH, pressure of carbon dioxide, pressure of oxygen, total carbon dioxide, bicarbonate, base excess, oxygen saturation, and lactate), the CG6+ cartridge (for sodium, potassium, chloride, blood glucose, blood urea nitrogen, hematocrit, and hemoglobin), and enzyme-linked immunosorbent assay kits (calcium, magnesium, creatinine, aspartate aminotransferase, alanine aminotransferase, bilirubin, and albumin). Similar trends were found for the parameters of biochemistries, electrolytes, and blood gas, and they revealed no significant changes after blood withdrawal-induced hemorrhagic shock. However, the TBS group showed more effective ability to correct metabolic acidosis than the NS and RS groups. TBS was a feasible and safe resuscitation solution in this study and may be an alternative to NS and RS for resuscitation in hemorrhagic shock patients without liver damage.

1985 related Products with: Comparison of the trometamol-balanced solution with two other crystalloid solutions for fluid resuscitation of a rat hemorrhagic model.

Human Ascites Fluid 25ml Hydrochloric Acid Soluti Phosphomolybdic Phosphot QuantiChrom™ Formaldehy Rat Anti-Mouse Forssman G Trypan Purple™ *0.1 M a N-(2-Amino-4,6-dichloro-5 Sheep Anti-Theophylline 3 Amplite™ Fluorimetric G Acetic Acid Solution (0. MOUSE ANTI BOVINE ROTAVIR FDA Standard Frozen Tissu

Related Pathways

  •  
  • No related Items
paperclip

#31939396   // Save this To Up

Status of adult immunity to hepatitis A virus in healthcare workers from a tertiary care hospital in north India.

Humans are considered to be the principal host for hepatitis A virus (HAV) infection. In India, heterogeneous groups of susceptible individuals coexist in different regions. There has been a decline in antibody titres to HAV among young adults which may pose a major public health problem. The objective of this study was to assess the IgG anti-HAV level among healthcare workers (HCWs) in the age group of 20-60 yr and its association with the socio-demographic variables.

2869 related Products with: Status of adult immunity to hepatitis A virus in healthcare workers from a tertiary care hospital in north India.

FIV Core Ag, recombinant Recombinant Hemagglutinin FDA Standard Frozen Tissu Advanced Airway Intubatio Mouse Anti-Influenza B Vi Rabbit Anti-Influenza A V Mouse Anti-Influenza A Vi Avian Influenza virus H5N Anti-Infectious Pancreati Mouse Anti-Influenza B Vi Mouse AntiInfluenza B Nuc Head & Neck cancer test t

Related Pathways

paperclip

#31935243   // Save this To Up

PR3 levels are impaired in plasma and PBMCs from Arabs with cardiovascular diseases.

Cardiovascular disease (CVD) risks persist in patients despite treatment. CVD susceptibility also varies with sex and ethnicity and is not entirely explained by conventional CVD risk factors. The aim of the present study was to identify novel CVD candidate markers in circulating Peripheral blood mononuclear cells (PBMCs) and plasma from Arab obese subjects with and without CVD using proteomic approaches. Human adults with confirmed CVD (n = 208) and matched non-CVD controls (n = 152) living in Kuwait were examined in the present cross-sectional study. Anthropometric and classical biochemical parameters were determined. We employed a shotgun proteomic profiling approach on PBMCs isolated from a subset of the groups (n = 4, each), and differentially expressed proteins selected between the two groups were validated at the mRNA level using RT-PCR (n = 6, each). Plasma levels of selected proteins from the proteomics profiling: Proteinase-3 (PR3), Annexin-A3 (ANX3), Defensin (DEFA1), and Matrix Metalloproteinase-9 (MMP9), were measured in the entire cohort using human enzyme-linked immunosorbent assay kits and were subsequently correlated with various clinical parameters. Out of the 1407 we identified and quantified from the proteomics profiling, 47 proteins were dysregulated with at least twofold change between the two subject groups. Among the differentially expressed proteins, 11 were confirmed at the mRNA levels. CVD influenced the levels of the shortlisted proteins (MMP9, PR3, ANX3, and DEFA1) in the PBMCs and plasma differentially. Despite the decreased levels of both protein and mRNA in PBMCs, PR3 circulating levels increased significantly in patients with CVD and were influenced by neither diabetes nor statin treatment. No significant changes were; however, observed in the DEFA1, MMP9, and ANX3 levels in plasma. Multivariate logistic regression analysis revealed that only PR3 was independently associated with CVD. Our results suggest that the dysregulation of PR3 levels in plasma and PBMCs reflects underlying residual CVD risks even in the treated population. More prospective and larger studies are required to establish the role of PR3 in CVD progression.

1161 related Products with: PR3 levels are impaired in plasma and PBMCs from Arabs with cardiovascular diseases.

Tissue array of gastric d Rabbit Plasma US Origin I Rabbit Plasma US Origin I HBV-3 panel test, HBsAg H Bovine Androstenedione,AS Infection diseases: Heli Biocidal ZF, spray disinf Rabbit Plasma US Origin I Rabbit Plasma US Origin I Prolactin-Inducible Prote Anti-HBcAg (HBcAb) test s Rabbit Plasma US Origin I

Related Pathways