Search results for: Mouse Anti-Progesterone
#25648617 2015/05/12 Save this To Up
Effect of Xianziyizhen Recipe Capsule on PGI2-PPARδ Signaling Pathway in Embryo Implantation Dysfunction Mice.We investigated the effect of Xianziyizhen recipe capsule (XRC), a kidney-tonifying herb, on the PGI2-PPARδ signaling pathway at the maternal-fetal interface in embryo implantation dysfunction (EID) mice.
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#22607890 2012/07/03 Save this To Up
Animal models of polycystic ovary syndrome: a focused review of rodent models in relationship to clinical phenotypes and cardiometabolic risk.To review rodent animal models of polycystic ovary syndrome (PCOS), with a focus on those associated with the metabolic syndrome and cardiovascular disease risk factors.
2889 related Products with: Animal models of polycystic ovary syndrome: a focused review of rodent models in relationship to clinical phenotypes and cardiometabolic risk.Ovary cancer tissue array Oral squamous cell cancer Ovary disease spectrum (o Ovary cancer survey tissu Ovary cancer survey tissu Ovary cancer survey tissu Ovary cancer survey tissu Ovary cancer survey tissu Ovary cancer and adjacent Ovary cancer tissue array Ovary cancer tissue array Ovary tumor tissue array,
#21417747 2011/04/13 Save this To Up
A case of progesterone-induced anaphylaxis, cyclic urticaria/angioedema, and autoimmune dermatitis.Women have exhibited anaphylaxis, urticaria/angioedema, and autoimmune progesterone dermatitis (APD) coinciding with the progesterone premenstrual rise. We report a detailed immunological evaluation of such a woman responsive to a gonadotropin hormone-releasing agonist (GHRA).
1872 related Products with: A case of progesterone-induced anaphylaxis, cyclic urticaria/angioedema, and autoimmune dermatitis.Anti AGO2 Human, Monoclon Anti AGO2 Mouse, Monoclon Anti AGO2 Human, Monoclon Anti AGO2 Mouse, Monoclon Confocal Dish,PS,clear, 3 Progesterone Receptor (Ph Androgen Receptor (Phosph Androgen Receptor (Phosph Rabbit Anti-Human Androge Rabbit Anti-Human Androge Progesterone Receptor (Ab Androgen Receptor (Ab 650
#18382274 2008/04/02 Save this To Up
Neuron-specific and inducible recombination by Cre recombinase in the mouse.To investigate the neuronal function of genes in vivo, a neuron-specific and inducible gene targeting system is desirable. In this study, we generated a knockin mouse line that expresses a fusion protein consisting of the Cre recombinase and the progesterone receptor (CrePR) in neurons. The neuron-specific expression of CrePR was attained by inserting CrePR gene into the tau locus, because tau is expressed strongly in neurons but scarcely in glias and other tissues. By crossing this knockin mouse line (tau(CrePR)) with ROSA26 lacZ reporter mouse line (R26R), we observed that the antiprogesterone RU486 could induce recombinase activity of the CrePR specifically in neurons. Thus, tau (CrePR) knockin line is a useful tool for studying neuronal gene functions.
2656 related Products with: Neuron-specific and inducible recombination by Cre recombinase in the mouse.BYL-719 Mechanisms: PI3K- CREB Phospho-Specific Arr Angiogenesis (Mouse) Anti Cytokine (Mouse) Antibody Cytokine (Mouse) Antibody Cytokine (Mouse) Antibody Cytokine (Mouse) Antibody Cytokine (Mouse) Antibody Inflammation (Mouse) Anti Angiogenesis (Mouse) Anti Atherosclerosis (Mouse) A Cytokine (Mouse) Antibody
#18197009 2008/01/16 Save this To Up
Promotion of BRCA1-associated triple-negative breast cancer by ovarian hormones.Mammary epithelial proliferation is controlled by the ovarian hormones estrogen and progesterone. Although BRCA1 (breast cancer 1, early onset) is ubiquitously expressed, women with BRCA1 mutations have a propensity to develop tumors in tissues sensitive to ovarian hormone. An understanding of the tissue-specific function of the BRCA1-encoded protein (BRCA1) provides additional insight that may improve cancer risk reduction in BRCA1 mutation carriers.
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#17618756 2007/07/09 Save this To Up
Arrest of preterm labor in rat and mouse by an oral and selective nonprostanoid antagonist of the prostaglandin F2alpha receptor (FP).The purpose of this study was to assess the tocolytic effect of AS604872, an orally active, potent, and selective prostanoid prostaglandin F2alpha receptor (FP) antagonist.
2222 related Products with: Arrest of preterm labor in rat and mouse by an oral and selective nonprostanoid antagonist of the prostaglandin F2alpha receptor (FP).AZD-3514 Mechanisms: Andr CAR,Car,Constitutive andr CAR,Car,Constitutive andr Goat Anti-Human, Mouse, R Goat Anti-Rat CCKA Recept Rat anti mouse CD119 (INF Rat monoclonal anti mouse Androgen Receptor , Mouse Androgen Receptor , Mouse Androgen Receptor (Phosph Androgen Receptor (Phosph Goat Anti-Mouse, Rat DLL1
#17143245 2006/12/04 Save this To Up
Calcium channel antagonists augment hydroxyurea- and ru486-induced inhibition of meningioma growth in vivo and in vitro.Although the chemotherapy drug hydroxyurea (HU) and the antiprogesterone mifepristone (RU486) have been used to treat meningiomas for which surgical and radiation therapies have failed, results have been disappointing. The addition of calcium channel antagonists (CCAs) to chemotherapeutic drugs enhances tumor growth inhibition in other tumor types, and the authors demonstrated that CCAs can block meningioma growth in vitro and in vivo. The purpose of this study was to test the effects of the addition of a CCA to HU or RU486 on meningioma growth.
2313 related Products with: Calcium channel antagonists augment hydroxyurea- and ru486-induced inhibition of meningioma growth in vivo and in vitro.Directed In Vivo Angiogen Anti AGO2 Human, Monoclon Anti AGO2 Mouse, Monoclon Anti AGO2 Human, Monoclon Anti AGO2 Mouse, Monoclon Human Insulin-like Growth Human Epstein-Barr Virus Human Insulin-like Growth Jurkat Cell Extract (Indu Jurkat Cell Extract (Indu Jurkat Cell Extract (Indu Jurkat Cell Extract (Indu
#17138902 2006/12/01 Save this To Up
Prevention of Brca1-mediated mammary tumorigenesis in mice by a progesterone antagonist.Women with mutations in the breast cancer susceptibility gene BRCA1 are predisposed to breast and ovarian cancers. Why the BRCA1 protein suppresses tumor development specifically in ovarian hormone-sensitive tissues remains unclear. We demonstrate that mammary glands of nulliparous Brca1/p53-deficient mice accumulate lateral branches and undergo extensive alveologenesis, a phenotype that occurs only during pregnancy in wild-type mice. Progesterone receptors, but not estrogen receptors, are overexpressed in the mutant mammary epithelial cells because of a defect in their degradation by the proteasome pathway. Treatment of Brca1/p53-deficient mice with the progesterone antagonist mifepristone (RU 486) prevented mammary tumorigenesis. These findings reveal a tissue-specific function for the BRCA1 protein and raise the possibility that antiprogesterone treatment may be useful for breast cancer prevention in individuals with BRCA1 mutations.
2980 related Products with: Prevention of Brca1-mediated mammary tumorigenesis in mice by a progesterone antagonist.BYL-719 Mechanisms: PI3K- Human Internal Mammary Ar GFP Expressing Human Inte Interleukins Recombinant Interleukins Recombinant Interleukins Recombinant Rat monoclonal anti mouse Interleukin-34 IL34 (N-t Interleukin-34 IL34 anti ING1B antisense AKT1 (dn) Inducible HIV 1 intergase antigen.
#16759651 2006/06/20 Save this To Up
Progesterone receptor antagonist is effective in relieving neuropathic pain.Injury to the spinal nerves of mice induces allodynia and hyperalgesia. Intrathecal injection of the progesterone/estrogen receptor antagonist ICI 182,780 produced antinociceptive effects. Co-administration of estrogen did not reduce but tended to enhance the antinociceptive effect of ICI 182,780. On the other hand, co-administration of progesterone dose-dependently reduced the antinociceptive effect of ICI 182,780, indicating that the antinociceptive effect is through antiprogesterone receptor activity of ICI 182,780. These results suggest that spinal progesterone receptors play an important role in neuropathic pain, and that controlling the activity of progesterone receptors may be of great importance in the treatment of neuropathic pain.
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#16129958 2005/08/30 Save this To Up
Progesterone-dependent immunomodulation.The biological effects of progesterone are mediated by a 34-kDa protein named the progesterone-induced blocking factor (PIBF). PIBF, synthesized by lymphocytes of healthy pregnant women in the presence of progesterone, inhibits arachidonic acid release as well as NK activity, and modifies the cytokine balance. Within the cell the full-length PIBF is associated with the centrosome, while secretion of shorter forms is induced by activation of the cell. PIBF induces nuclear translocation of STAT6 as well as PKC phosphorylation and exerts a negative effect on STAT4 phosphorylation. The concentration of PIBF in pregnancy urine is related to the positive or negative outcome of pregnancy; furthermore, premature pregnancy termination is predictable by lower than normal pregnancy PIBF values. In vivo data suggest the biological importance of the above findings. Treatment of pregnant Balb/c mice with the antiprogesterone RU 486 results in an increased resorption rate, which is associated with the inability of spleen cells to produce PIBF. High resorption rates induced by progesterone receptor block as well as those due to high NK activity are corrected by simultaneous PIBF treatment.
Progesterone Receptor (P Progesterone Receptor (P Progesterone Receptor (P Malic enzyme 2, NAD(+) de Progesterone Receptor (Ph Progesterone Receptor (Ab PABP1-dependent poly A-sp voltage-dependent calcium Progesterone receptor ant Steroid AccuBind™ ELISA Rabbit Anti-IEX1 Differen Rabbit Anti-IEX1 Differen
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