Search results for: Rabbit Anti-Human CD290 Antibodies





Inflammatory cell expression of Toll-like receptor-2 (TLR2) within refractory periapical granuloma.
Toll-like receptor-2 (TLR2) is highly important within the immune system. Characterization of the expression of TLR2 within inflammatory cells in periapical lesions could help in diagnosis and management of refractory cases. The aim of the study is identification of Toll-like receptor (TLR2) through immunohistochemical and immunofluroscence expression in inflammatory cells within refractory periapical granuloma cases. Eight cases of refractory periapical granuloma were selected out of 772 cases. Histological examination and immunohistochemical staining with polyclonal rabbit antihuman TLR2, monoclonal mouse antihuman CD38, CD68 and CD83 primary antibodies, as well as immunofluorescence staining with goat anti-rabbit TLR2, donkey anti-mouse CD38, CD68 and CD83 primary antibodies was conducted. Positive controls, negative controls and experimental sections with no primary antibody were included in the study. Qualitative analysis and double immunofluorescence technique was used to characterize the TLR cells. In periapical granuloma, lymphocytes (CD38 cells) expressed the most amount of TLR reactivity followed by macrophages (CD68 cells), and odontogenic epithelial cells. Neutrophils, red blood cells (RBCs) and collagen ground substance were negative to TLR2. TLR2 was highly expressed by lymphocytes and plasma cells indicative of their major role in the inflammatory process and antigen recognition in refractory periapical granuloma. Dendritic cells expressing TLR2 were low in number suggesting a minor role in sustaining these lesions.
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Human Killer cell immunog
Rat Toll Like Receptor 2(
Rat monoclonal anti mouse
Cellufine GCL-2000 Media
Pressure Injection Cell w
Adenosine A2b Receptor
Androgen Receptor Ab-1 An
Rabbit Anti-Cell death in
Bradykinin receptor B1 An
Activin receptor-like kin
Rabbit Anti-Human Toll-Li
Glucagon receptor
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Immunohistochemical expression of polo-like kinase 1 in oral squamous cell carcinoma and oral submucous fibrosis.
Polo-like kinase 1 (PLK1) is a critical molecule in the proliferation of several human cancers. Overexpression of PLK1 has been correlated with cancer cell proliferation and lower overall survival rates. Although PLK1 has been studied in various tumors, information regarding its expression in oral cancer and precancer is limited. Aims: This study is aimed at evaluating the expression of PLK1 in a potentially malignant and malignant disorder of the oral cavity, namely, oral submucous fibrosis (OSMF) and oral squamous cell carcinoma (OSCC), respectively, using the immunohistochemistry technique. It also intended to evaluate the association of the various histological grades of OSCC with the intensity of PLK1 expression.
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Oral cavity squamous cell
Oral squamous cell cancer
Cervix squamous cell carc
Esophageal squamous cell
Skin squamous cell carcin
Esophagus squamous cell c
Esophageal squamous cell
Esophagus squamous cell c
Lung squamous cell carcin
Esophagus squamous cell c
Multiple organ squamous c
Lung squamous cell carcin
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Posttransplant Cyclophosphamide for HLA-haploidentical Transplantation in Patients With Mucopolysaccharidosis.
We successfully used a haploidentical transplantation protocol with posttransplant cyclophosphamide (CY) (50 mg/kg/d on days +3 and +4) for in vivo T-cell depletion in patients with mucopolysaccharidosis using reduced-intensive conditioning regimens, followed by a busulfan-based conditioning regimen, which included busulfan (12 to 16 mg/kg) and fludarabine(150 to 200 mg/m)+rabbit antihuman thymocyte globulin (7.5 to 10 mg/kg) as a conditioning regimen. Cyclosporine or tacrolimus, methotrexate, mycophenolate mofetil, and methylprednisolone were administered to prevent graft-versus-host disease (GVHD). After follow-up for a median period of 1.5 years, all 8 patients without preexisting severe comorbidities and early transplant referrals are alive, with 100% donor chimerism and excellent performance status. Only 1 patient developed chronic GVHD(II). We conclude that posttransplant CY is effective in vivo for T-cell depletion to promote full donor engraftment in patients with mucopolysaccharidosis. In addition, with posttransplant CY, the procedure reduced the rate of GVHD and the cost of transplant and improved the patients' quality of life.
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Multiple lung carcinoma (
Indole 7 carboxaldehyde (
Syringe pump can be contr
Breast invasive ductal ca
Indole 6 carboxaldehyde (
Indole 4 carboxaldehyde (
Indole 3 carboxaldehyde (
(7’-Benzyloxy-indolymet
Indole 5 carboxaldehyde (
Breast cancer tissue arra
Internexin NF66 Antibody,
Lung cancer test tissue a
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Induction of Antihuman C-C Chemokine Receptor Type 5 Antibodies by a Bovine Herpesvirus Type-4 Based Vector.
Bovine herpesvirus 4 (BoHV-4) is a promising vector for the delivery and intracellular expression of recombinant antigens and can thus be considered as a new prototype vaccine formulation system. An interesting, and actively pursued, antigen in the context of human immunodeficiency virus (HIV) infection prophylaxis (and therapy) is the C-C chemokine receptor type 5 (CCR5) co-receptor, whose blockage by specific antibodies has been shown to inhibit both viral entry and cell-to-cell transmission of the virus. Building on our previous work on the BoHV-4 vector system, we have engineered and tested a replication-competent derivative of BoHV-4 (BoHV-4-CMV-hCCR5ΔTK) bearing a human CCR5 (hCCR5) expression cassette. We show here that CCR5 is indeed expressed at high levels in multiple types of BoHV-4-CMV-hCCR5ΔTK-infected cells. More importantly, two intravenous inoculations of CCR5-expressing BoHV-4 virions into rabbits led to the production of anti-CCR5 antibodies capable of reacting with the CCR5 receptor exposed on the surface of HEK293T cells through specific recognition of the amino-terminal region (aa 14-34) of the protein. Given the growing interest for anti-CCR5 immunization as an HIV control strategy and the many advantages of virus-based immunogen formulations (especially for poorly immunogenic or self-antigens), the results reported in this study provide preliminary validation of BoHV-4 as a safe viral vector suitable for CCR5 vaccination.
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Rabbit Anti-Human Cholecy
Rabbit Anti-Human Cholecy
Herpesvirus Type 1 (BHV 1
TNFRSF1B - Goat polyclona
CGKIT 2 Collagenase assay
Mouse anti-bovine type II
Mouse anti-bovine type II
ELHGBII Human Monkey IgG
MOUSE ANTI BOVINE ROTAVIR
Rabbit Anti-Chicken Colla
Rabbit Anti-C. botulinum
ELMGBI Mouse IgG anti bov
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Development and Validation of an Enzyme-Linked Immunosorbent Assay for the Detection of Binding Anti-Drug Antibodies against Interferon Beta.
To develop and validate a method for the detection of binding anti-drug antibodies (ADAs) against interferon beta (IFN-β) in human serum as part of a European initiative (ABIRISK) aimed at the prediction and analysis of clinical relevance of anti-biopharmaceutical immunization to minimize the risk.
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Rabbit Anti-Rat Androgen
Rat monoclonal anti mouse
Rat Anti-Mouse beta-Gluca
Rabbit Anti-Rat GRO-beta
Mouse Anti-IL-1 beta Anti
Goat Anti-Human Cdc42-bin
Rabbit Anti-Rat IL-1 beta
Mouse Anti-Spectrin Bindi
Rabbit Anti-Rat GABA A Re
Mouse anti human NRG1 bet
Rabbit Anti-Mouse Amyloid
FDA Standard Frozen Tissu
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Anti-Gal and Anti-Neu5Gc Responses in Nonimmunosuppressed Patients After Treatment With Rabbit Antithymocyte Polyclonal IgGs.
Polyclonal antihuman thymocyte rabbit IgGs (antithymocyte globulin [ATG]) are popular immunosuppressive drugs used to prevent or treat organ or bone-marrow allograft rejection, graft versus host disease, and autoimmune diseases. However, animal-derived glycoproteins are also strongly immunogenic and rabbit ATG induces serum sickness disease in almost all patients without additional immunosuppressive drugs, as seen in the Study of Thymoglobulin to arrest Type 1 Diabetes (START) trial of ATG therapy in new-onset type 1 diabetes.
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Rabbit Anti-IAA (Indole-3
Rabbit Anti-ING1 p33 Poly
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Rabbit Anti-Integrin beta
Rabbit Anti-Integrin alph
Rabbit Anti-Integrin alph
Rabbit Anti-galectin 9 Po
Rabbit Anti-Integrin alph
Rabbit Anti-APIP Apaf1 In
Rabbit Anti-galectin 9 Po
Rabbit Anti-intestinal FA
Rabbit Anti-Insulin Recep
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Electrochemical Sandwich Immunosensor for Determination of Exosomes Based on Surface Marker-Mediated Signal Amplification.
Extracellular vesicles (EVs), namely, exosomes and microvesicles, are important mediators of intercellular communication pathways. Since EVs can be detected in a variety of biofluids and contain a specific set of biomarkers which are reminiscent of their parental cells, they show great promise in clinical diagnostics as EV analysis can be performed in minimally invasive liquid biopsies. However, reliable, fast and cost-effective methods for their determination are still needed, especially if decentralized analysis is intended. In this study, we developed an electrochemical biosensor which works with 1.5 μL sample volume and can detect as low as 200 exosomes per microliter, with a linear range spanning almost 4 orders of magnitude. The sensor is specific and readily differentiates exosomes from microvesicles in samples containing 1000-fold excess of the latter. Capability of detecting exosomes in real samples (diluted serum) was shown. This was achieved by immobilizing rabbit antihuman CD9 antibodies on gold substrates and using monoclonal antibodies against CD9 for detection of captured exosomes. Signal amplification is presumably obtained from the fact that multiple detector antibodies bind to the surface of each captured vesicle. Detection is performed based on electrochemical reduction of 3,3',5,5'-tetramethyl benzidine (TMB) after addition of horseradish peroxidase (HRP)-conjugated anti-IgG antibodies. This amperometric biosensor can be easily incorporated into future miniaturized and semiautomatic devices for EV determination.
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QuantiChromâ„¢ LDH Cytoto
QuantiChromâ„¢ Formaldehy
MarkerGeneTM Fluorescent
Anti-Hepatitis B Surface
MOUSE ANTI HUMAN CD15, Pr
Rabbit Anti-gamma tubulin
Recombinant Hepatitis B S
Rabbit PAI-1 (wild type l
Hepatitis B surface Ag (H
formin-like 1 antibody So
Rabbit Anti-gamma tubulin
QuantiChromâ„¢ Urea Assay
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An Analytical Comparison of Dako 28-8 PharmDx Assay and an E1L3N Laboratory-Developed Test in the Immunohistochemical Detection of Programmed Death-Ligand 1.
Nivolumab, a fully human immunoglobulin G4 programmed death-1 (PD-1) immune checkpoint inhibitor antibody, has activity in melanoma, non-small-cell lung cancer (NSCLC), renal cell carcinoma (RCC), and Hodgkin lymphoma. Nivolumab is approved in the USA and EU for advanced melanoma, NSCLC, and RCC, and relapsed Hodgkin lymphoma in the USA. Programmed death-ligand 1 (PD-L1), a PD-1 ligand, is expressed on mononuclear leukocytes, myeloid cells, and tumor cells. PD-L1 is being investigated as a potential biomarker to predict the association of tumor PD-L1 expression with nivolumab efficacy.
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Cultrex In Vitro Angiogen
Alkaline Phospatase (ALP)
Multiple organ tumor tiss
Breast cancer test tissue
HCV antibody test strip,
H. Pylori antibody test s
Rabbit Anti-Cell death in
Her-2 IHC Bio-marker cont
Rabbit Anti-Cell death in
Esophageal cancer test ti
Annexin V PE Cy5 Apoptosi
FDA Standard Frozen Tissu
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Immunosuppressive therapy for kidney transplantation in children and adolescents: systematic review and economic evaluation.
End-stage renal disease is a long-term irreversible decline in kidney function requiring kidney transplantation, haemodialysis or peritoneal dialysis. The preferred option is kidney transplantation followed by induction and maintenance immunosuppressive therapy to reduce the risk of kidney rejection and prolong graft survival.
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17β-Acetoxy-2α-bromo-5Î
Androstenedione-19 Antibo
Androgen Receptor (Ab 650
Indole 4 carboxaldehyde (
Androgen Receptor (Ab 650
Anti Androgen Receptor pr
Kidney cancer tissue arra
Androgen Receptor Antibod
High density kidney cance
Androst-16-en-3-ol C19H30
Androsta-3,5,16-trien-17-
Kidney clear cell carcino
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Preemptive CD20+ B cell Depletion Attenuates Cardiac Allograft Vasculopathy in CD154-Treated Monkeys.
Anti-CD154 monotherapy is associated with antidonor allo-antibody (Ab) elaboration, cardiac allograft vasculopathy (CAV), and allograft failure in preclinical primate cell and organ transplant models. In the context of calcineurin inhibitors (CNI), these pathogenic phenomena are delayed by preemptive "induction" B cell depletion.
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CELLKINES Natural Human I
Rabbit Anti-Cell death in
Leptin ELISA Kit, Rat Lep
Biocidal ZF, spray disinf
baculoGROW insect cell me
Biocidal ZF, spray disinf
Alamar Blueâ„¢, REDOX ind
MarkerGeneTM in vivo lacZ
Octyl â D 1 thioglucopyr
Nile Red, A lipophilic dy
Biocidal ZF, spray disinf
Alamar Blueâ„¢, REDOX ind
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