Search results for: Recombinant Human IL-16 [+His] Proteins
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[Recombinant human IL-16 inhibits proliferation and induces apoptosis in MT-4 cells].To investigate the effects of recombinant human IL-16 (rhIL-16) on the proliferation and apoptosis of MT-4 cells.
1658 related Products with: [Recombinant human IL-16 inhibits proliferation and induces apoptosis in MT-4 cells].Macrophage Colony Stimula Macrophage Colony Stimula Recombinant Human Interfe Recombinant Human Interle Human Internal Mammary Ar Recombinant Human Interle Recombinant Human Interle Recombinant Human Insulin Recombinant Human HGF [fr Recombinant Human Interle Recombinant Human p16-INK Recombinant Human PEDF [f
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Increased expression of S100 calcium binding protein A8 in GM-CSF-stimulated neutrophils leads to the increased expressions of IL-8 and IL-16.In our previous proteomic surveillance, we found that at least 11 proteins in neutrophils were increased more than 2.5-fold by the stimulation of GM-CSF. In this paper, focusing on one of the 11 proteins, S100 calcium binding protein A8 (S100A8), we tried to elucidate the effect of S100A8 and the cooperative effect of S100A8 and GM-CSF on production and secretion of cytokines of neutrophils.
2990 related Products with: Increased expression of S100 calcium binding protein A8 in GM-CSF-stimulated neutrophils leads to the increased expressions of IL-8 and IL-16.ELISA kit Calcyclin,Chick Human S100 Calcium Bindin Recombinant Human S100B C Chicken S100 calcium bind S100 alpha - Rabbit polyc FDA Standard Frozen Tissu Ofloxacin CAS Number [824 Rabbit Anti-Rat Androgen Rat monoclonal anti mouse Goat Anti-Human Vitamin D FIV Core Ag, recombinant Rabbit Anti-Human Toll In
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Down-regulation of interleukin-16 in human mast cells HMC-1 by Clostridium difficile toxins A and B.Toxin A (TcdA) and toxin B (TcdB) are the major virulence factors of Clostridium difficile and are the causative agents for clinical symptoms, such as secretory diarrhoea and pseudomembranous colitis. Mast cells are essentially involved in the toxin-induced colonic inflammatory processes. To study the direct effects of these toxins on the expression of inflammatory genes, a DNA microarray containing evaluated probes of 90 selected inflammatory genes was applied to the immature mast cell line HMC-1. TcdA and TcdB induced up-regulation of only a limited number of genes within the early phase of cell treatment. Interleukin-8 (IL-8), transcription factor c-jun and heme oxygenase-1 messenger RNA (mRNA) increased more than 2-fold. In contrast, IL-16, known as a CD4(+) T-cell chemoattractant factor and the chemokine receptor cKit were down-regulated. Stimulation of HMC-1 cells with IL-8 had no effect on IL-16 mRNA level, indicating that both cytokines were independently affected by the toxins. Regulation of both cytokines, however, depended on glucosylation of Rho GTPases as tested by application of enzyme-deficient TcdA or TcdB. Down-regulation of total and secreted IL-16 protein was checked by enzyme-linked immunosorbent assay. The data implicate that TcdA and TcdB affect lymphocyte migration by modulating release of the chemoattractant factor IL-16 from mast cells. In addition, this is the first report showing that Rho GTPases are involved in the regulation of IL-16 expression.
2309 related Products with: Down-regulation of interleukin-16 in human mast cells HMC-1 by Clostridium difficile toxins A and B.Interleukin-24 antibody S interleukin 17 receptor C Rabbit Anti-APIP Apaf1 In Mouse Anti-Human Interleu Hamster anti mouse Interl Mouse Anti-Human Interleu Human Interleukin-17AF He Indole 5 carboxylic acid Inhibitory mouse monoclo Anti-human C1 Esterase In Rabbit Anti-Human ATM (aa Mouse Anti-Human Interleu
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The inhibitor profiling of the caspase family of proteases using substrate-derived peptide glyoxals.A series of substrate-based α-keto-β-aldehyde (glyoxal) sequences have been synthesised and evaluated as inhibitors of the caspase family of cysteine proteases. A number of potent inhibitor sequences have been identified. For example, a palmitic acid containing sequence pal-Tyr-Val-Ala-Asp-glyoxal was demonstrated to be an extremely effective inhibitor of caspase-1, inhibiting not only the action of the protease against synthetic fluorogenic substrates (K(i)=0.3 nM) but also blocking its processing of pro-interleukin-1beta (pro-IL-1β). In addition, the peptide Ac-Asp-Glu-Val-Asp-glyoxal, which is based on the consensus cleavage sequence for caspase-3, is a potent inhibitor of this protease (K(i)=0.26 nM) yet only functions as a comparatively modest inhibitor of caspase-1 (K(i)=451 nM). Potent inhibitor sequences were also identified for caspases-6 and -8. However, the degree of discrimination between the family members is limited. The ability of Ac-Asp-Glu-Val-Asp-glyoxal to block caspase-3 like activity in whole cells and to delay the development of apoptosis was assessed. When tested against caspase-3 like activity in cell lysates, Ac-Asp-Glu-Val-Asp-glyoxal displayed effective inhibition similar to that observed against recombinant caspase-3. Treatment of whole cells with this potent caspase-3 inhibitor was however, not sufficient to significantly stall the development of apoptosis in-vitro.
1777 related Products with: The inhibitor profiling of the caspase family of proteases using substrate-derived peptide glyoxals.Caspase Family Inhibitor Caspase Family Inhibitor Caspase Family Inhibitor Caspase-Family Inhibitor Caspase-Family Inhibitor Caspase Family Inhibitor Caspase Family Inhibitor Pfu DNA Polymerase protei Caspase Family Inhibitor Caspase Family Inhibitor Caspase-Family Inhibitor Caspase Family Inhibitor
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[Serum cytokine profiling of prostate cancer and benign prostatic hyperplasia using recombinant antibody microarray].To identify the differential expressions of serum cytokines between prostate cancer (PCa) and benign prostatic hyperplasia (BPH), and provide proteomic evidence for the early diagnosis of PCa.
1554 related Products with: [Serum cytokine profiling of prostate cancer and benign prostatic hyperplasia using recombinant antibody microarray].Anti-Human Benign Prostat Anti-Human Benign Prostat Prostate cancer, PIN (pro Prostate cancer, hyperpla Cancer samples: Prostate Prostate cancer, hyperpla Prostate cancer and hyper Prostate disease spectrum Top 4 types of cancer (co Prostate cancer and hyper Prostate cancer, hyperpla Tissue microarray of top
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Interleukin-16 inhibits immunoglobulin e production by B lymphocytes.The increased production of IgE is a hallmark of atopic disorders. CD4+ T cells regulate the production of Immunoglobulin (Ig) E by B cells. Interleukin (IL) 16, a CD4+ specific cytokine, is highly expressed at sites of allergic inflammation. Our aim was to determine the effect of IL-16 on IgE production in atopic subjects.
3-O-Benzyl Estrone Monoet Horizontal Laminar Flow 2-[5-[2-[4-(1,2-Benzisoth AccuPrep Genomic DNA Extr Animal Biologicals Immun Horizontal Laminar Flow 5-[2-[4-(1,2-Benzisothiaz (S)-(+)-Benzyl Glycidyl E 5-Bromoindole-2-carboxyli Vertical Laminar Flow Cle 4’-O-Benzyloxy Ezetimib Human Interleukin 1 Beta
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