Search results for: Triglyceride Quantification Kit
#25855471 2015/11/30 Save this To Up
2,3-Oxidosqualene cyclase protects liver cells from the injury of intermittent hypoxia by regulating lipid metabolism.2,3-Oxidosqualene cyclase (OSC), an important enzyme of cholesterol biosynthesis, catalyzes the highly selective cyclization of 2,3-monoepoxysqualene to lanosterol. Intermittent hypoxia (IH) is a hallmark feature in obstructive sleep apnea (OSA) which is increasingly recognized as an independent risk factor for liver injury. The aim of this study was to determine the effect of IH on OSC expression and evaluate the role of OSC in the IH-induced apoptosis in hepatic cell line human liver cell (HL-02).
2731 related Products with: 2,3-Oxidosqualene cyclase protects liver cells from the injury of intermittent hypoxia by regulating lipid metabolism.Leptin ELISA Kit, Rat Lep Human Liver Sinusoidal Mi GFP Expressing Human Live RFP Expressing Human Live Nile Red, A lipophilic dy Fontana-Masson Stain Kit Fontana-Masson Stain Kit Guanylate Cyclase β1 Carnitine O palmitoyltran Anti C Reactive Protein A anti HSV (II) gB IgG1 (mo anti HCMV IE pp65 IgG1 (m
#25096952 2014/09/02 Save this To Up
Fatty acid binding protein-4 is expressed in the mouse placental labyrinth, yet is dispensable for placental triglyceride accumulation and fetal growth.Fatty Acid Binding Protein-4 (FABP4) is a member of a family of FABP proteins that regulate intracellular lipid trafficking in diverse tissues. We recently showed that FABP4 regulates triglyceride accumulation in primary human trophoblasts. To assess the function of placental FABP4 in vivo, we tested the hypothesis that FABP4 is expressed in the murine placenta, and regulates placenta triglyceride accumulation.
2470 related Products with: Fatty acid binding protein-4 is expressed in the mouse placental labyrinth, yet is dispensable for placental triglyceride accumulation and fetal growth.Rabbit Anti-IAA (Indole-3 Rabbit Anti-APIP Apaf1 In Rabbit Anti-APIP Apaf1 In Rabbit Anti-APIP Apaf1 In Rabbit Anti-TNIP2 ABIN2 T Rabbit Anti-TNIP2 ABIN2 T Rabbit Anti-TNIP2 ABIN2 T Rabbit Anti-Cell death in Rabbit Anti-Cell death in Rabbit Anti-Cell death in Rabbit Anti-P2RX4 ATP gat Rabbit Anti-P2RX4 ATP gat
#24768786 2014/06/02 Save this To Up
Analytical performances of a new enzymatic assay for hemoglobin A1c.HbA1c is considered the gold standard for the follow-up of diabetic patients and a new diagnostic tool for diabetes mellitus, which implies the availability of reliable assay methods. We have evaluated a new assay developed by Abbott Laboratories, based on the enzymatic quantification of HbA1c by a fructosyl dipeptide oxidase using Architect analyzers.
Hemoglobin A1c antibody, Hemoglobin A1c antibody, Native Human Hemoglobin A Native Human Hemoglobin A Native Human Hemoglobin A Glucose Assay With the La Cultrex In Vitro Angiogen ENZYMATIC ASSAY KITS (CH Potassium Ion Assay (Enzy Sodium Ion Assay (Enzymat ENZYMATIC ASSAY KITS (CH ENZYMATIC ASSAY KITS (CH
#22505639 2012/06/18 Save this To Up
Mast cell deficiency attenuates progression of atherosclerosis and hepatic steatosis in apolipoprotein E-null mice.Mast cells are important cells of the immune system and are recognized as participants in the pathogenesis of atherosclerosis. In this study, we evaluated the role of mast cells on the progression of atherosclerosis and hepatic steatosis using the apolipoprotein E-deficient (ApoE(-/-)) and ApoE(-/-)/mast cell-deficient (Kit(W-sh/W-sh)) mouse models maintained on a high-fat diet. The en face analyses of aortas showed a marked reduction in plaque coverage in ApoE(-/-)/Kit(W-sh/W-sh) compared with ApoE(-/-) after a 6-mo regimen with no significant change noted after 3 mo. Quantification of intima/media thickness on hematoxylin and eosin-stained histological cross sections of the aortic arch revealed no significant difference between ApoE(-/-) and ApoE(-/-)/Kit(W-sh/W-sh) mice. The high-fat regimen did not induce atherosclerosis in either Kit(W-sh/W-sh) or wild-type mice. Mast cells with indications of degranulation were seen only in the aortic walls and heart of ApoE(-/-) mice. Compared with ApoE(-/-) mice, the serum levels of total cholesterol, low-density lipoprotein and high-density lipoprotein were decreased by 50% in ApoE(-/-)/Kit(W-sh/W-sh) mice, whereas no appreciable differences were noted in serum levels of triglycerides or very low density lipoprotein. ApoE(-/-)/Kit(W-sh/W-sh) mice developed significantly less hepatic steatosis than ApoE(-/-) mice after the 3-mo regimen. The analysis of Th1/Th2/Th17 cytokine profile in the sera revealed significant reduction of interleukin (IL)-6 and IL-10 in ApoE(-/-)/Kit(W-sh/W-sh) mice compared with ApoE(-/-) mice. The assessment of systemic generation of thromboxane A(2) (TXA(2)) and prostaglandin I(2) (PGI(2)) revealed significant decrease in the production of PGI(2) in ApoE(-/-)/Kit(W-sh/W-sh) mice with no change in TXA(2). The decrease in PGI(2) production was found to be associated with reduced levels of cyclooxygenase-2 mRNA in the aortic tissues. A significant reduction in T-lymphocytes and macrophages was noted in the atheromas of the ApoE(-/-)/Kit(W-sh/W-sh) mice. These results demonstrate the direct involvement of mast cells in the progression of atherosclerosis and hepatic steatosis.
1377 related Products with: Mast cell deficiency attenuates progression of atherosclerosis and hepatic steatosis in apolipoprotein E-null mice.Eosinophil - Mast Cell S Anti C Reactive Protein A anti HSV (II) gB IgG1 (mo anti HCMV IE pp65 IgG1 (m anti HCMV gB IgG1 (monocl CELLKINES Natural Human I CELLKINES INTERLEUKIN 2 ( CELLKINES INTERLEUKIN 2 ( Jurkat Cell Extract (Indu Jurkat Cell Extract (Indu Jurkat Cell Extract (Indu Jurkat Cell Extract (Indu
#21063085 2010/11/24 Save this To Up
Blood arachidonic acid and HDL cholesterol influence the phagocytic abilities of human monocytes/macrophages.Many immunomodulators may intensify the process of phagocytosis in monocytes. Among them are the fatty acids contained in cellular membrane phospholipids. But in the available literature there are no reports on how blood fatty acids and lipoproteins can modulate the phagocytic abilities of cells. Therefore, the aim of this study was the evaluation of the phagocytic activity of monocytes isolated from the blood of healthy human subjects with defined fatty acids and lipoprotein content.
1450 related Products with: Blood arachidonic acid and HDL cholesterol influence the phagocytic abilities of human monocytes/macrophages.Fibroblast Growth Factor Fibroblast Growth Factor Human Fibroblast Growth F Rabbit Anti-Human Androge Rabbit Anti-Human Androge anti A1, A2 human blood a Macrophage Colony Stimula anti A1, A2, A3 human blo anti B human blood antige anti AB human blood antig Macrophage Colony Stimula Blood Group Antibodies a
#15047469 2004/03/29 Save this To Up
[Value of non-esterified fatty acids quantification in diabetes].Non-esterified fatty acids (NEFA) play a complex role in glucidic homeostasis. This role led us to investigate their quantification in diabetic patients. The plasmatic NEFA concentrations, measured with the FA115 kit of Randox, showed significant differences between control patients (0.42 +/- 0.14 mmol/L, N = 50) and diabetic patients (0.68 +/- 0.35 mmol/L, p < 0.01, 443 diabetic patients (70 with type l and 373 with type 2 diabetes)). NEFA concentrations were significantly higher in type 2 diabetics (0.70 +/- 0.32 mmol/L) when compared to type 1 diabetics (0.59 +/- 0.35 mmol/L, p < 0.05). In type 2 diabetics, a significant correlation was observed between NEFA and glucose concentrations at 8 hrs a.m., and the mean glucose concentrations along the day (p < 0.001). In contrast NEFA concentrations were less correlated to levels of HbA1c. NEFA were well correlated with cholesterol and triglycerides (p < 0.05) but not with Lp(a). They were also correlated with BMI but not with age or duration of the disease. Diabetic patients on metformin associated to lipolytic treatment, presented lower concentrations of NEFA and better glucidic control. The results confirm the role of NEFA in glucidic homeostasis and suggest an interest for their routine determination.
Triglyceride Assay Kit Li Leptin ELISA Kit, Rat Lep Anti 3 DG imidazolone Mon Rat intestinal fatty acid Free Fatty Acid Quantific Interleukin-34 IL34 (N-t Interleukin-34 IL34 anti Fatty acid free heat sho Fatty acid free heat sho Fatty acid free heat sho Fatty acid free heat sho Sterile filtered goat se
#12875241 2003/07/23 Save this To Up
[Studies on the antigenic stability of hepatic triglyceride lipase in human postheparin plasma during long-term storage].The present study describes how to process human postheparin plasma (PHP) containing hepatic triglyceride lipase (HTGL) that is utilized as a standard material of HTGL for the quantification of HTGL mass in human plasma. The optimal storage conditions for PHP were established by monitoring the stability of HTGL molecules in PHP as an antigen, which was stored in the liquid, frozen, or lyophilized state, using purified human PHP-HTGL as the standard material and a commercial HTGL ELISA MARUPI kit, which is a direct sandwich enzyme-linked immunosorbent assay (ELISA). The HTGL ELISA MARUPI kit, for which the validity was confirmed by precision and dilution tests, showed that the immunoreactive mass of HTGL in lyophilized PHP remained stable for at least 12 months at a storage temperature of 4 degrees C or lower. These results indicate that lyophilized PHP stored at a temperature of less than 4 degrees C can be utilized as the standard material for the quantification of HTGL in human plasma using the HTGL ELISA MARUPI kit.
2937 related Products with: [Studies on the antigenic stability of hepatic triglyceride lipase in human postheparin plasma during long-term storage].Goat Anti-Human Monoglyce Goat Anti-Human HMGB3 HMG Goat Anti-Human F2R PAR1, Goat Anti-Human Endotheli Lipoproteins, Human Plasm Human interleukin 2(IL-2) Goat Anti-Human ABCE1 RNA Goat Anti-Human ACOX2, (i Goat Anti-Human AOC3, (in Goat Anti-Human APOA4, (i Goat Anti-Human Bruno-lik Goat Anti-Human CACNB4 (i
#10074887 1999/05/21 Save this To Up
Development and evaluation of a direct sandwich enzyme-linked immunosorbent assay for the quantification of lipoprotein lipase mass in human plasma.The purpose of this study was to develop and evaluate a direct sandwich enzyme-linked immunosorbent assay (ELISA) for quantification of the lipoprotein lipase (LPL) immunoreactive mass in human plasma using monoclonal antibodies (MAbs) directed against LPL purified from human postheparin plasma (PHP) [corrected].
2487 related Products with: Development and evaluation of a direct sandwich enzyme-linked immunosorbent assay for the quantification of lipoprotein lipase mass in human plasma.Leptin ELISA Kit, Human A Goat Anti-Human Endotheli Alkaline Phospatase (ALP) Directed In Vivo Angiogen Lipoproteins, Human Plasm Human interleukin 2(IL-2) Goat Anti-Human Monoglyce Mouse Anti-Insulin-Like G Mouse Anti-Lipoprotein Li Anti AGO2 Human, Monoclon Anti AGO2 Human, Monoclon Bone Morphogenetic Protei
#8327993 1993/08/06 Save this To Up
Factor VII activation and oral contraceptives.
Factor VIII Related Anti Factor VIII Related Anti Factor VIII Related Anti Sheep Anti-Human Factor V Recombinant Human Factor Recombinant Human Factor Recombinant Human Factor Recombinant Human Factor Recombinant Human Factor Recombinant Human Factor Native Human Coagulation Native Human Coagulation
#3779953 1986/12/29 Save this To Up
Interference from lipemia in a radioimmunoassay kit for quantification of choriogonadotropin.
1368 related Products with: Interference from lipemia in a radioimmunoassay kit for quantification of choriogonadotropin.Amplite™ Fluorimetric F Amplite™ Fluorimetric H Amplite™ Intracellular Amplite™ Fluorimetric P Amplite™ Fluorimetric A EtBr Destaining Bag Kit A Cell Meter™ Intracellul Cell Meter™ Fluorimetri Cell Meter™ Fluorimetri Beta Amyloid (42) ELISA K GLP 1 ELISA Kit, Rat Gluc Beta Amyloid (1 40) ELISA
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