Search results for: MBPmyelin
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[Effect of PP60c-Src on angiotensin II-induced signal transduction in rat vascular smooth muscle cells].
To further clarify the mechanism of Ang II-induced intracellular signal transduction in vascular smooth muscle cells(VSMCs) proliferation by observing the effect of c-Src on Ang II-mediated MAPK activation and c-fos protein expressions in rat VSMCs.Mei-xiang Xiang, Dong Liu, Peng Chen, Jun-Sheng Lu
2788 related Products with: [Effect of PP60c-Src on angiotensin II-induced signal transduction in rat vascular smooth muscle cells].
2 Pieces/Box2 Pieces/Box100ul0.5 ml1.00 flask1x10e7 cells1 mg100ug Lyophilized2 Pieces/Box100ul
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CEK2, a Novel MAPK from Candida albicans Complement the Mating Defect of fus3/kss1 Mutant.
A novel MAPK(mitogen-activated protein kinase) gene, CEK2(Candida albicans extracellular signal-regulated kinase 2), was isolated by screening the Candida albicans library based on oligonucleotide probe hybridization and degenerated PCR. The CEK2 gene is 1 119 bp in length, and coding for a 373 aa protein. The CEK2 shares 56% homology with CEK1 from Candida albicans, 55 % homology with FUS3 from S. cerevisiae. From the deduced amino acid sequence, the Cek2 protein contains a conserved ATP binding site and a Ser/Thr kinase activity signal and a conserved TEY sequence was located at L(12) region. In vitro kinase activity assay showed Cek2 could phosphorylate MBP(myelin bovine protein) but not histone H1. CEK2 gene could complement the fus3/kss1 mutant and underwent mating signal induction by a tester strain, but CEK1 gene could not complement with the fus3/kss1 mutant. CEK2 is therefore a FUS3 homolog in Candida albicans.Jing Chen, Qing Wang, Jiang-Ye Chen
2943 related Products with: CEK2, a Novel MAPK from Candida albicans Complement the Mating Defect of fus3/kss1 Mutant.
100 ug1 mL1 mg1 mL1 mL100ug100ug Lyophilized100 µg100 ul100ug100ug Lyophilized
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Chromosome 19 single-locus and multilocus haplotype associations with multiple sclerosis. Evidence of a new susceptibility locus in Caucasian and Chinese patients.
Susceptibility to multiple sclerosis (MS) involves a genetically complex autoimmune component. However, except for genes in the HLA system, specific susceptibility loci are unknown or unconfirmed.L F Barcellos, G Thomson, M Carrington, J Schafer, A B Begovich, P Lin, X H Xu, B Q Min, D Marti, W Klitz